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Precision Redox Platform
PROTECT-SOD™
A diagnostic and tagatose-based therapeutic platform for cardiometabolic and brain health, guided by precision genetics.
New Executive Summary
Many common chronic diseases are driven by oxidative stress — an imbalance between damaging molecules and the body's ability to neutralize them. Some people are born with gene variants that weaken their natural antioxidant defenses, putting them at higher lifetime risk for heart disease, diabetes, dementia, and certain neurodevelopmental disorders. PROTECT-SOD identifies these high-risk individuals and treats them with a tagatose-centered nutraceutical program.
Precision Genetics
We identify SOD2, GPX1, and APOE gene variants that weaken natural antioxidant defenses.
Redox Balance
We restore the balance between damaging molecules and the body's ability to neutralize them.
Cardiometabolic Health
We reduce the risk of heart disease, diabetes, metabolic syndrome, and insulin resistance.
Brain Health
We address cognitive decline, dementia, and neurodevelopmental conditions through early intervention.
The Science
Biology in Simple Terms
What is SOD2?
Superoxide Dismutase 2 (SOD2) is a key antioxidant enzyme located inside mitochondria. It converts superoxide into less harmful molecules, acting as a first-line defense against oxidative stress.
SOD2 TT Genotype
Individuals with the TT genotype have less efficient SOD2 transport into mitochondria, resulting in higher oxidative stress. We also examine GPX1 and APOE to refine the risk profile.
At-Risk Population
Adults with ≥2 cardiometabolic risk factors — obesity, hypertension, type 2 diabetes, dyslipidemia — combined with high-risk SOD2/GPX1/APOE genetics create a double-hit susceptibility.
Neurodegeneration & Neurodevelopment
In Alzheimer's and subsets of autism, oxidative stress, mitochondrial dysfunction, and gut–brain axis disruptions play central roles. PROTECT-SOD addresses these shared biological pathways.
Intervention
Why Tagatose?
Tagatose is a naturally occurring "rare sugar" that tastes and functions like regular sugar but behaves very differently metabolically. This combination — low glycemic load + gut-microbiome modulation — makes tagatose ideal as the core scaffold of a longevity-oriented nutraceutical.
Low Glycemic Impact
Fewer digestible calories and an ultra-low impact on blood glucose and insulin levels.
Microbiome Modulation
Favors beneficial gut bacteria and increases short-chain fatty acids like butyrate.
Daily Formulation
Daily oral gummy with Fibersol-2, pectin, and marine algae components designed for long-term adherence.
Clinical Study
Study Overview
~300
Participants
18–85
Age Range (years)
3–6
Months of Intervention
Cleveland Clinic Florida · Prospective Study · SOD2, GPX1, APOE Genetic Testing
AIM 01
Map Genetic Vulnerability
Quantify how common SOD2-TT and related high-risk genotypes are in a real-world cardiometabolic clinic and how they correlate with baseline oxidative imbalance.
AIM 02
Test Tagatose-Based Correction
Determine whether our tagatose-centered formulation improves SOD2 activity, glutathione, butyrate, and related markers over 3–6 months.
AIM 03
Link Biology to Health
Assess how biomarker improvements relate to changes in metabolic risk factors, symptoms, and patient-reported quality of life.
Products
From Study to Platform Product
Diagnostics Product
A clinically deployable panel (SOD2/GPX1/APOE + redox/microbiome biomarkers) for cardiology, neurology, and longevity centers.
Tagatose-Based Therapeutics
A family of targeted formulations — cardio-metabolic, brain-focused, pediatric/neurodevelopmental — anchored on tagatose plus co-factors tuned to genotype.
Software & Data
Redox risk scores and response algorithms to guide whom to test, treat, and monitor — licensable as digital decision-support tools.
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